Full report by Berenis - Swiss Expert Group on electromagnetic fields and non-ionizing radiation
by David Schuermann, Department of Biomedicine, University of Basel, Mattenstrasse 28, CH-4058 Basel, Switzerland and
Meike Mevissen, Veterinary Pharmacology and Toxicology, Vetsuisse Faculty, University of Bern, Laenggassstrasse 124, CH-3012 Bern, Switzerland
′′ A trend is emerging, where it becomes clear that even when you consider methodological weaknesses, EMF exposure, even in the low dose interval, can very well give changes in cellular oxidative balance."
‘In summary, indications for increased oxidative stress caused by RF-EMF and ELF-MF were reported in the majority of the animal studies and in more than half of the cell studies. Investigations in Wistar and Sprague-Dawley rats provided consistent evidence for oxidative stress occurring after RF-EMF exposure in the brain and testes and some indication of oxidative stress in the heart. Observations in Sprague-Dawley rats also seem to provide consistent evidence for oxidative stress in the liver and kidneys. In mice, oxidative stress induced by RF-EMF was predominantly demonstrated in the brain and testes, as well as in liver, kidneys, and ovaries. These observations were made with a variety of cell types, exposure times, and dosages (SAR or field strengths), within the range of the regulatory limits and recommendations.
Certainly, some studies were subject to methodological uncertainties or weaknesses or are not very comprehensive regarding exposure time, dose, number, and quantitative analysis of the biomarkers used, to name a few. A trend is emerging, which becomes clear even when taking these methodological weaknesses into account, i.e., that EMF exposure, even in the low dose range, may well lead to changes in cellular oxidative balance. Organisms and cells are able to react to oxidative stress, and many observations after EMF exposure point to an adaptation after a recovery phase. Adverse conditions, such as diseases (diabetes, neurodegenerative diseases), compromise the body’s defense mechanisms, including antioxidant protection mechanisms, and individuals with such pre-existing conditions are more likely to experience health effects. The studies show that very young or old individuals can react less efficiently to oxidative stress, which of course also applies to other stressors that cause oxidative stress. Further investigations under standardized conditions are necessary to better understand and confirm these phenomena and observations.’
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